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    Tu ne sais pas combien elle est belle

    Dong, Xi, 1966-
    Darras, Isild
    La Tour-d'Aigues : Ed. de l'Aube
    2013
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    Title: Tu ne sais pas combien elle est belle / Dong Xi ; nouvelles traduites du chinois par Isild Darras
    Author: Dong, Xi, 1966-
    Contributor: Darras, Isild
    Publisher: La Tour-d'Aigues : Ed. de l'Aube
    Date: 2013
    Format: 231 p.
    Series: L'Aube poche
    Documents in this series: L'Aube poche
    Identifier: 9782815907125 (ISBN); http://catalogue.bnf.fr/ark:/12148/cb43533815b (URN)
    RERO No.: R007251126
    Permalink:
    http://data.rero.ch/01-R007251126/html

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    Sauver une vie

    Dong, Xi, 1966-
    Manon, Amélie
    La Tour-d'Aigues : Ed. de l'Aube
    2013
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    Title: Sauver une vie / Dong Xi ; court roman trad. du chinois par Amélie Manon
    Author: Dong, Xi, 1966-
    Contributor: Manon, Amélie
    Publisher: La Tour-d'Aigues : Ed. de l'Aube
    Date: 2013
    Format: 139 p.
    Series: Regards croisés
    Documents in this series: Regards croisés
    Note: Traduit de: Juiming
    Identifier: 9782815907095 (ISBN); http://catalogue.bnf.fr/ark:/12148/cb435341896 (URN)
    RERO No.: R007251135
    Permalink:
    http://data.rero.ch/01-R007251135/html

    • Several versions

    Holographic entanglement entropy for general higher derivative gravity

    Xi Dong
    Journal of High Energy Physics, 10 January 2014, Vol.1401 [Peer Reviewed Journal]

    • Several versions

    Association of 677 C>T (rs1801133) and 1298 A>C (rs1801131) polymorphisms in the MTHFR gene and breast cancer susceptibility: a meta-analysis based on 57 individual studies

    Kai Li, Wusheng Li, Xi Dong
    PloS one, 01 January 2014, Vol.9(6), p.e71290 [Peer Reviewed Journal]

    • Several versions

    Combined Adenovirus-Mediated Artificial microRNAs Targeting mfgl2, mFas, and mTNFR1 Protect against Fulminant Hepatic Failure in Mice (Virus-Mediated miRNA Treatment of Hepatic Failure)

    Xi, Dong, Wang, Ming, Ye, Huali, Luo, Xiaoping, Ning, Qin
    2013, Vol.8(11), p.e82330 [Peer Reviewed Journal]

    • Several versions

    Ablation of Sim1 Neurons Causes Obesity through Hyperphagia and Reduced Energy Expenditure (Obesity Due to Sim1 Neuron Ablation)

    Xi, Dong, Gandhi, Nilay, Lai, Meizan, Kublaoui, Bassil M
    PLoS ONE, 2012, Vol.7(4), p.e36453 [Peer Reviewed Journal]

    • Several versions

    Paraventricular nucleus Sim1 neuron ablation mediated obesity is resistant to high fat diet

    Xi, Dong, Roizen, Jeff, Lai, Meizan, Gandhi, Nilay, Kublaoui, Bassil
    PloS one, 2013, Vol.8(11), pp.e81087 [Peer Reviewed Journal]

    • Several versions

    Correction: Paraventricular Nucleus Sim1 Neuron Ablation Mediated Obesity Is Resistant to High Fat Diet

    Xi, Dong, Roizen, Jeff, Lai, Meizan, Gandhi, Nilay, Kublaoui, Bassil
    PloS one, 2014, Vol.9(1) [Peer Reviewed Journal]

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    Phosphoinositide kinases play key roles in norepinephrine- and angiotensin II-induced increase in phosphatidylinositol 4,5-bisphosphate and modulation of cardiac function

    Xu, Jia-Xi, Si, Man, Zhang, Hui-Ran, Chen, Xing-Juan, Zhang, Xi-Dong, Wang, Chuan, Du, Xiao-Na, Zhang, Hai-Lin
    The Journal of biological chemistry, 07 March 2014, Vol.289(10), pp.6941-8 [Peer Reviewed Journal]
    MEDLINE/PubMed (U.S. National Library of Medicine)
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    Title: Phosphoinositide kinases play key roles in norepinephrine- and angiotensin II-induced increase in phosphatidylinositol 4,5-bisphosphate and modulation of cardiac function
    Author: Xu, Jia-Xi; Si, Man; Zhang, Hui-Ran; Chen, Xing-Juan; Zhang, Xi-Dong; Wang, Chuan; Du, Xiao-Na; Zhang, Hai-Lin
    Subject: Cardiac Hypertrophy ; G Protein-Coupled Receptors (Gpcr) ; Phosphatidylinositol Kinase ; Phospholipid Metabolism ; Signal Transduction ; Angiotensin II -- Physiology ; Cardiomegaly -- Physiopathology ; Norepinephrine -- Physiology ; Phosphatidylinositol 4,5-Diphosphate -- Metabolism ; Phosphatidylinositol-4-Phosphate 3-Kinase -- Physiology
    Description: The seemly paradoxical Gq agonist-stimulated phosphoinositide production has long been known, but the underlying mechanism and its physiological significance are not known. In this study, we studied cardiac phosphoinositide levels in both cells and whole animals under the stimulation of norepinephrine (NE), angiotensin II (Ang II), and other physiologically relevant interventions. The results demonstrated that activation of membrane receptors related to NE or Ang II caused an initial increase and a later fall in phosphatidylinositol 4,5-bisphosphate (PIP2) levels in the primary cultured cardiomyocytes from adult rats. The possible mechanism underlying this increase in PIP2 was found to be through an enhanced activity of phosphatidylinositol 4-kinase IIIβ, which was mediated by an up-regulated interaction between phosphatidylinositol 4-kinase IIIβ and PKC; the increased activity of phosphatidylinositol 4-phosphate 5-kinase γ was also involved for NE-induced increase of PIP2. When the systolic...
    Is part of: The Journal of biological chemistry, 07 March 2014, Vol.289(10), pp.6941-8
    Identifier: 1083-351X (E-ISSN); 24448808 Version (PMID); 10.1074/jbc.M113.527952 (DOI)

    • Several versions

    Entropy, extremality, euclidean variations, and the equations of motion

    Xi Dong, Aitor Lewkowycz
    The journal of high energy physics, 01 January 2018, Vol.2018(1), pp.1-33 [Peer Reviewed Journal]