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    L'île au bout du corridor : [roman]

    Liaudet, Marie Antoinette
    Paris : Talleyrand
    2006
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    Titre: L'île au bout du corridor : [roman] / Marie Antoinette Liaudet
    Auteur: Liaudet, Marie Antoinette
    Editeur: Paris : Talleyrand
    Date: 2006
    Collation: 183 p.
    Description: Roman de société
    Valaisanne habitant Yverdon
    Décrit dans: Migros Magazine, éd. Valais, 2007,18,p.6-9 (Alain Portner et Jean-François Duval)
    Classification: vs-bvs-sys 09.01.01
    cdu-vsmart 82-3 LIAU
    Identifiant: 2353370063 (ISBN); http://catalogue.bnf.fr/ark:/12148/cb40957315m (URN)
    No RERO: R004322270
    Permalien:
    http://data.rero.ch/01-R004322270/html?view=VS_V1

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    Nonselective versus Selective Inhibition of Inducible Nitric Oxide Synthase in Experimental Endotoxic Shock

    Liaudet, Lucas
    Rosselet, Anne, Schaller, Marie-Denise, Markert, Michèle, Perret, Claude, Feihl, François
    Journal of Infectious Diseases. - 1998/177/1/127-132
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    Titre: Nonselective versus Selective Inhibition of Inducible Nitric Oxide Synthase in Experimental Endotoxic Shock
    Auteur: Liaudet, Lucas
    Contributeur: Rosselet, Anne; Schaller, Marie-Denise; Markert, Michèle; Perret, Claude; Feihl, François
    Description: The effects of two nitric oxide synthase (NOS) inhibitors with different isoform selectivity were compared in a murine model of endotoxemia. Mice challenged with 70 mg/kg intraperitoneal (ip) lipopolysaccharide (LPS) were treated 6 h after LPS with either NG-γ-L-arginine methyl ester (LNAME, nonselective NOS inhibitor, 10-60 mg/kg), L-canavanine (selective inhibitor of inducible NOS, 50-300 mg/kg), or saline (0.2 mL) given ip. In a subset of mice, plasma concentrations of nitrate (NO breakdown product), lipase (pancreas injury), lactate dehydrogenase, and transaminases (liver injury) were measured 16 h after LPS. Although both inhibitors reduced plasma nitrate, they produced contrasting effects on survival and organ injury. L-NAME enhanced liver damage and tended to accelerate the time of death, while L-canavanine significantly reduced mortality and had no deleterious effects in terms of organ damage. These results indicate that nonselective NOS inhibitors are detrimental in endotoxic shock and support the potential usefulness of selective inducible NOS inhibitors in this setting
    Publication en relation: Journal of Infectious Diseases. - 1998/177/1/127-132
    Document hôte: Journal of Infectious Diseases
    Identifiant: 10.1086/513813 (DOI)

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    Epidemiology, diagnosis, and treatment of pulmonary embolism

    Turini, Pierre, Schaller, Marie-Denise, Liaudet, Lucas
    Revue medicale de la Suisse romande, June 2004, Vol.124(6), pp.351-7 [Revue évaluée par les pairs]
    MEDLINE/PubMed (U.S. National Library of Medicine)
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    Titre: Epidemiology, diagnosis, and treatment of pulmonary embolism
    Auteur: Turini, Pierre; Schaller, Marie-Denise; Liaudet, Lucas
    Sujet: Pulmonary Embolism
    Description: Pulmonary embolism (PE) is a frequent pathology, the mortality of which remains elevated despite the efficacy of anticoagulation. This is mainly due to diagnostic difficulty, resulting from the low sensibility and specificity of clinical signs and routine exams in PE. The best diagnostic approach relies on decision making algorithms based on the determination of a clinical probability, which in turn dictates the choice of specialized exams (C-scan/scintigraphy, venous duplex, angiography and D-dimers). Risk stratification, which is based on the presence of hemodynamic alterations (right ventricular dysfunction, shock) is essential to guide therapy. In case of shock (massive embolism), treatment relies on systemic thrombolysis. In the presence of right ventricular dysfunction without shock (sub-massive embolism), the advantage of thrombolysis over anticoagulation alone has not been clearly demonstrated. In all other cases, as long as no contraindication exists, therapeutic anticoagulation...
    Fait partie de: Revue medicale de la Suisse romande, June 2004, Vol.124(6), pp.351-7
    Identifiant: 0035-3655 (ISSN); 15293444 Version (PMID)

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    Peroxynitrite is a potent inhibitor of NF-{kappa}B activation triggered by inflammatory stimuli in cardiac and endothelial cell lines

    Levrand, Sandra, Pesse, Benoît, Feihl, François, Waeber, Bernard, Pacher, Pal, Rolli, Joëlle, Schaller, Marie-Denise, Liaudet, Lucas
    The Journal of biological chemistry, 14 October 2005, Vol.280(41), pp.34878-87 [Revue évaluée par les pairs]
    MEDLINE/PubMed (U.S. National Library of Medicine)
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    Titre: Peroxynitrite is a potent inhibitor of NF-{kappa}B activation triggered by inflammatory stimuli in cardiac and endothelial cell lines
    Auteur: Levrand, Sandra; Pesse, Benoît; Feihl, François; Waeber, Bernard; Pacher, Pal; Rolli, Joëlle; Schaller, Marie-Denise; Liaudet, Lucas
    Sujet: Endothelial Cells -- Cytology ; Myocardium -- Cytology ; Nf-Kappa B -- Chemistry ; Peroxynitrous Acid -- Pharmacology
    Description: Peroxynitrite is a potent oxidant and nitrating species proposed as a direct effector of myocardial damage in numerous cardiac pathologies. Whether peroxynitrite also acts indirectly, by modulating cell signal transduction in the myocardium, has not been investigated. Therefore, we examined a possible role for peroxynitrite on the activation of NF-kappaB, a crucial pro-inflammatory transcription factor, in cultured H9C2 cardiomyocytes. H9C2 cells were stimulated with tumor necrosis factor-alpha or lipopolysaccharide following a brief (20-min) exposure to peroxynitrite. NF-kappaB activation (phosphorylation and degradation of its inhibitor IkappaBalpha, nuclear translocation of NF-kappaB p65, and NF-kappaB DNA binding) triggered by lipopolysaccharide or tumor necrosis factor-alpha was abrogated by peroxynitrite. Peroxynitrite also inhibited NF-kappaB in two human endothelial cell lines activated with tumor necrosis factor-alpha or interleukin-1beta. These effects were related to oxidative...
    Fait partie de: The Journal of biological chemistry, 14 October 2005, Vol.280(41), pp.34878-87
    Identifiant: 0021-9258 (ISSN); 16079150 Version (PMID)

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    New therapeutic strategies in severe sepsis and septic shock

    Oddo, Mauro, Schaller, Marie-Denise, Calandra, Thierry, Liaudet, Lucas
    Revue medicale de la Suisse romande, June 2004, Vol.124(6), pp.329-32 [Revue évaluée par les pairs]
    MEDLINE/PubMed (U.S. National Library of Medicine)
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    Titre: New therapeutic strategies in severe sepsis and septic shock
    Auteur: Oddo, Mauro; Schaller, Marie-Denise; Calandra, Thierry; Liaudet, Lucas
    Sujet: Sepsis -- Therapy ; Shock, Septic -- Therapy
    Description: Severe sepsis and septic shock are an important cause of mortality. Until recently, in spite of major progresses in our understanding of the pathogenic mechanisms of this syndrome, clinicians had only a limited therapeutic arsenal. Considerable efforts have been made in the past few years to develop novel therapeutic interventions to reduce mortality in sepsis. So far, five specific therapies have proven their efficacy to achieve such goal in large randomised controlled trials: early goal-directed therapy, recombinant activated protein C, moderate doses of steroids, low tidal volume ventilation in acute respiratory distress syndrome and intensive insulin therapy to control hyperglycemia. This review will focus on these recently acquired therapeutic modalities, that are presently available to clinicians for the treatment of severe sepsis and septic shock.
    Fait partie de: Revue medicale de la Suisse romande, June 2004, Vol.124(6), pp.329-32
    Identifiant: 0035-3655 (ISSN); 15293439 Version (PMID)

    • Plusieurs versions

    Catalytically inactive human cathepsin D triggers fibroblast invasive growth

    Laurent-Matha, Valérie, Maruani-Herrmann, Sharon, Prébois, Christine, Beaujouin, Mélanie, Glondu, Murielle, Noël, Agnès, Alvarez-Gonzalez, Marie Luz, Blacher, Sylvia, Coopman, Peter, Baghdiguian, Stephen, Gilles, Christine, Loncarek, Jadranka, Freiss, Gilles, Vignon, Françoise
    The Journal of cell biology, 31 January 2005, Vol.168(3), pp.489-99 [Revue évaluée par les pairs]

    • Plusieurs versions

    Comparison of skin microvascular reactivity with hemostatic markers of endothelial dysfunction and damage in type 2 diabetes

    Beer, Sandra, Feihl, François, Ruiz, Juan, Juhan-Vague, Irène, Aillaud, Marie-Françoise, Wetzel, Sandrine Golay, Liaudet, Lucas, Gaillard, Rolf C, Waeber, Bernard
    Vascular Health and Risk Management, 2008, Vol.4(6), p.1449-1458 [Revue évaluée par les pairs]

    • Plusieurs versions

    Impact of standardized calibration on the inter-assay variation of 14 automated assays for the measurement of creatinine in human serum

    Séronie-Vivien, Sophie, Galteau, Marie-Madeleine, Carlier, Marie-Christine, Hadj-Aissa, Aoumeur, Hanser, Anne-Marie, Hym, Bernadette, Marchal, Alain, Michotey, Odile, Pouteil-Noble, Claire, Sternberg, Michel, Perret-Liaudet, Armand
    Clinical chemistry and laboratory medicine, 2005, Vol.43(11), pp.1227-33 [Revue évaluée par les pairs]

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    Endothelium-dependent vasodilation in the skin microcirculation of patients with septic shock

    Kubli, Sandrine, Boëgli, Yann, Ave, Anne Dalle, Liaudet, Lucas, Revelly, Jean-Pierre, Golay, Sandrine, Broccard, Alain, Waeber, Bernard, Schaller, Marie-Denise, Feihl, François
    Shock (Augusta, Ga.), March 2003, Vol.19(3), pp.274-80 [Revue évaluée par les pairs]
    MEDLINE/PubMed (U.S. National Library of Medicine)
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    Titre: Endothelium-dependent vasodilation in the skin microcirculation of patients with septic shock
    Auteur: Kubli, Sandrine; Boëgli, Yann; Ave, Anne Dalle; Liaudet, Lucas; Revelly, Jean-Pierre; Golay, Sandrine; Broccard, Alain; Waeber, Bernard; Schaller, Marie-Denise; Feihl, François
    Sujet: Microcirculation -- Physiopathology ; Shock, Septic -- Physiopathology ; Skin -- Blood Supply ; Vasodilation -- Physiology
    Description: The evidence for endothelial dysfunction in sepsis is mostly restricted to animal models. We investigated endothelial function in the skin microcirculation of eight patients hospitalized for septic shock in an intensive care unit (ICU). All patients required adrenergic support. Twelve hemodynamically stable ICU patients without sepsis who did not receive any vasoactive medication were used as controls. The two groups were of similar age and sex ratio. For additional reference, 16 healthy, nonsmoking subjects matched for age and sex to the first two groups were also studied. The evaluation of endothelial function was based on the comparison of skin blood flow responses to iontophoretically applied acetylcholine (Ach, an endothelium-dependent vasodilator) and sodium nitroprusside (SNP, an endothelium-independent vasodilator). Skin blood flow was measured on the volar face of the forearm using laser Doppler imaging. Before application of Ach or SNP, the mean baseline skin blood flow was below...
    Fait partie de: Shock (Augusta, Ga.), March 2003, Vol.19(3), pp.274-80
    Identifiant: 1073-2322 (ISSN); 12630529 Version (PMID)

    • Plusieurs versions

    Beyond PrP res Type 1/Type 2 Dichotomy in Creutzfeldt-Jakob Disease (PrPres Diversity and s and i CJD)

    Uro-Coste, Emmanuelle, Cassard, Hervé, Simon, Stéphanie, Lugan, Séverine, Bilheude, Jean-Marc, Perret-Liaudet, Armand, Ironside, James W, Haik, Stéphane, Basset-Leobon, Christelle, Lacroux, Caroline, Peoch', Katell, Streichenberger, Nathalie, Langeveld, Jan, Head, Mark W, Grassi, Jacques, Hauw, Jean-Jacques, Schelcher, Francois, Delisle, Marie Bernadette, Andréoletti, Olivier
    PLoS Pathogens, 2008, Vol.4(3), p.e1000029 [Revue évaluée par les pairs]